Date of Submission

8-2024

Document Type

Thesis

Degree Name

Master of Science in Cellular and Molecular Biology

Department

Biology and Environmental Sciences

Advisor

Anna Kloc, Ph.D.

Committee Member

Amy Carlile, Ph.D.

Committee Member

Alireza G. Senejani, Ph.D.

Committee Member

Anthony Melillo, M.S

Keywords

Viral mRNA, Epstein-Barr Virus (EBV), Human Heart, Myocarditis, Cardiovascular Health

MeSH

RNA, Messenger, Herpesvirus 4, Human, Heart, Myocarditis

LCSH

Messenger RNA, Epstein-Barr virus, Epstein-Barr virus diseases, Heart--Biopsy, Myocarditis

Abstract

Myocarditis, characterized by inflammation of the myocardium, can lead to tissue necrosis and degeneration. While it shares similarities with other common illnesses, accurate diagnosis is crucial due to its potentially severe consequences. This study focuses on investigating the presence and reactivation of Epstein-Barr virus (EBV) in myocarditis by analyzing human heart tissue samples. Specifically, the study aims to detect EBV genome and EBV viral mRNA in human heart tissue samples using nucleic acid extraction, cDNA generation, and PCR analysis with specific primers targeting genes involved in EBV reactivation. Seventy-two heart tissue samples, previously collected from patients with myocarditis, dilated cardiomyopathy, or non-cardiovascular-related causes, were used in this study.

The extracted nucleic acid samples were screened for DNA contamination and treated with DNase enzyme. Finally, gel electrophoresis was used to visualize PCR-amplified bands, which were sequenced for EBV genome confirmation. The EBV was successfully identified in 4 out of the 72 analyzed samples. Only patients who had cardiovascular disease at the time of heart tissue samples collection had the viral genome in their hearts, in contrast to the control patient population. The study's findings provided valuable insights into the potential association of EBV genome presence in heart tissue and disease, which could have implications for cardiovascular health.

Available for download on Friday, August 17, 2029

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